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In patients with end-stage renal disease, because the kidneys cannot work properly, phosphorus cannot be eliminated and accumulated in the blood, resulting in hyperphosphatemia. The excretion of phosphorus ions is reduced, the phosphate radicals in the blood are increased, and the calcium ions are decreased. In addition, the production and metabolism of active vitamin D3 are hindered, and the absorption of calcium is problematic. Long-term calcium and phosphorus imbalances can lead to itchy skin, severe bone lesions, and tissue calcification.

In daily life, the intake of phosphorus-containing diet should be limited, and phosphorus-lowering drugs (phosphorus binders) should be used in conjunction to inhibit the absorption of phosphorus in the intestines, so as to achieve effective control and improve the quality of life.

Normal serum phosphate and calcium

• Normal serum phosphorus: 3.5-5.5mg/dL
• Normal serum calcium: 8.4-9.5mg/dL
• The product of calcium and phosphorus values: less than 55

Common causes of hyperphosphatemia

• Eating too many foods containing high phosphorus
• Phosphorus-lowering drugs (phosphorus binders) were taken the wrong way.
• I forgot to bring phosphorus-lowering drugs (phosphorus binders) when I went out to eat, so I didn't take them.
• Irregular dialysis (dialysis) or insufficient dialysis

Symptoms of elevated blood phosphorus

Bone headache, back pain, weakness of limbs, inability to climb stairs or sitting and need to rely on hands to stand, fractures, itchy skin, insomnia, anemia, etc., and more serious arrhythmia, cardiomyopathy, heart weakness , vascular sclerosis, etc.

Types of phosphate-lowering drugs and how to take them correctly